Analysis of Causes That Led to Baby Alan Ream Yurko’s
Cardiac Arrest and Death in November of 1997

Mohammed Ali Al-Bayati, PhD, DABT, DABVT
Toxicologist & Pathologist

maalbayati@toxi-health.com

http://www.toxi-health.com

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MAIN CONTENTS

List of Tables

PAGE 5 CONTENTS

Section
V. Review of Alan R.Yurko’s Jury Trial, & Analysis of Expert Witnesses’ Testimonies
and the State’s Claim

Introduction
A. Review of Dr. Ben Guedes’ testimony [click]
B. Review of Dr. Matthew A. Seibel’s testimony [click]
C. Review of Dr. Gary Steven Pearl’s testimony [click]
D. Review of Dr. Douglas R. Shanklin’s testimony [click]
E. Analysis of the state’s claim
[click]

Section VI. Conclusions and Recommendations [click]

References [click]


Section V. Review of Alan R.Yurko’s Jury Trial, & Analysis of the Expert Witnesses’ Testimonies and the State’s Claim

Introduction
Alan R. Yurko’s jury trial took place February 22 to 24, 1999 in the state of Florida [13]. The prosecutor provided four major witnesses testifying for the state, and (following the defense witness) two of these were called for repeat appearances before the jury. Against these four, the defense provided a single witness. The state witnesses were: 1) Dr. Shashi B. Gore, the medical Examiner; 2) Dr. Gary Pearl, a consultant neuropathologist (testified twice); 3) Dr. Ben Guedes, the treating physician; and 4) Dr. Matthew A. Seibel, a general pediatrician (testified twice). The defense witness was Dr. Douglas Radford Shanklin, a pathologist. In addition, two radiologists, Dr. James Edward Hannah and Dr. Robert Scott Mahan, testified in relation to the fractured rib #6; and Dr. Robert Gold, ophthalmologist, gave testimony in relation to the minor bleeding in the retina.

Three state witnesses (Gore, Guedes, and Seibel) stated that baby Alan died as the result of Shaken Baby Syndrome. However, none of them provided medical evidence to prove their case, and their testimonies were based only on a theory. Dr. Pearl stated that the injuries in the brain and spinal cord were acute injuries and did not start at birth or after birth. He did not say that these injuries were caused by Shaken Baby Syndrome. His findings are discussed in Section V-C, below.


In the previous section (IV) of this report is a review and analysis of the medical examiner autopsy report and his testimony in court relating to this case. They clearly show that Gore did not review all the medical evidence, and he did not prove his case. In addition, he described the histology of the heart, even though the heart had been donated for transplantation prior to his examination. Also, in court he stated that the cerebrospinal fluid was mixed with blood, but in his report he stated that the CSF was clear. In this section, I present my review and analysis of the testimonies of the other three state witnesses. This presentation shows that these witnesses also did not review all the evidence and did not provide the medical evidence to prove their case. I also present my review of the defense witness. He made important contributions to this case.

V-A. Review of Dr. Ben Guedes’ testimony
Dr. Ben Guedes stated in court that baby Alan died as a result of “Shaken Baby Syndrome.” His theory was that shaking the head caused bleeding, and that the blood pushed on the brain causing an increase in pressure. The pressure on the brain stem caused unconsciousness and cessation of breathing. Dr. Guedes presented his conclusion about the cause of death without making any attempt to review Francine’s medical record during her pregnancy with Alan, the baby’s medical records from birth until his hospitalization on November 24, 1997, the adverse reactions to vaccines and medications given to the baby, and the autopsy findings. I explained these issues and their impact on Alan’s health in sections I through IV of this report. It is very clear that Guedes’ conclusion about the cause of death was based on a theory and not on facts, because he did not evaluate the medical evidence related to this case.


Furthermore, Guedes presented in court only selected items of his clinical findings related to baby Alan’s five days in the Princeton and Florida hospitals. He did not reveal that, at the time of his admission to Princeton Hospital, Alan had diabetes and complications of diabetes, such as gastric ulcer, metabolic acidosis, elevated anion gap, dehydration, anemia, elevated serum liver enzymes and LDH (Table 12). Furthermore, Guedes did not reveal to the court that he treated baby Alan with a long list of medications that included three types of antibiotics, Tylenol, Motrin, heparin, antidiuretic hormones, potassium, and others
(Table 6, Table 7, Table 11). The clinical findings during Alan’s hospitalization on November 24th through 29th, along with an analysis of the clinical events are presented in Section III of this report. Below is a brief summary of these clinical findings to show that Guedes was aware that baby Alan had diabetes, hypokalemia, metabolic acidosis, infections, and dehydration, because he reviewed the data and treated him for these conditions. Unfortunately, his treatment with excessive doses of sodium bicarbonate and heparin caused severe hypoxia and severe bleeding in the brain, lungs, and spinal cord.


Dr. Guedes examined baby Alan at Princeton Hospital shortly after his admission. He saw the baby at about 12:30 PM on November 24, 1997 in the emergency room after the emergency department physicians resuscitated him. The baby arrived at Princeton with cardiac arrest and apnea. Dr. Guedes’ initial evaluation revealed that baby Alan was flaccid, his abdomen was soft and somewhat distended, and there was no bowel sound. The baby developed bleeding from the gastrostomy tube due to a gastric ulcer. His corneas were somewhat cloudy. Guedes also reviewed the result of the first laboratory work done at Princeton. The first blood gas was done at 12:09 PM showing a pH of 7.179, a PCO2 of 74 mm Hg, and a PO2 of 585 mm Hg
(Table 8). Other laboratory work drawn at 12:09 PM showed a glucose of 337 mg/dL, LDH of 2411 IU/L, SGOT of 207 IU/L, SGPT of 121 IU/L, a CO2 of 13 mEq/L, anion gap of 22 mEq/L, and potassium of 4.9 mEq/L. The white blood cell count was 20,900 per µL, hematocrit 25.3%, hemoglobin concentration 7.8 g/dL, and the platelet count was 571,000 per µL (Table 12).

These tests clearly showed that the baby suffered from diabetes, metabolic and respiratory acidosis, bacterial infections, anemia, and liver and heart damage. In Princeton Hospital, Dr. Guedes treated the baby with sodium bicarbonate to control acidosis; three types of antibiotics—rocephin, gentamicin, and Claforan (cefotaxime sodium)—to control bacterial infection; and saline to treat dehydration (Table 6). He also treated Alan in Florida Hospital on November 24 through November 28, 1997. He gave the baby antibiotics, Tylenol, Motrin, sodium bicarbonate, potassium chloride, heparin, dopamine, adenosine, muscle relaxant, antidiuretic hormone, and other medications (Table 7, Table 11). The baby responded very well to antibiotics. His temperature, white blood cell count, and blood glucose levels returned to normal following two days of treatment (Tables 8, 12, Table 13).


On November 26th, his serum glucose level dropped to a normal level of 95 mg/dL from the level of 397 mg/dL (76% reduction) on November 24, 1997. Also, on November 26th, the LDH, alkaline phosphate, and SGPT levels dropped by 70%, 47%, and 19% respectively from their levels on November 24. On 11/26, the total white blood cell count was reduced by 35% from its level on November 24th. This clearly indicates that the baby had liver and pancreas bacterial infections, and that his infections were resolved because of the treatment with antibiotics
(Table 6, Table 7).


Unfortunately, Dr. Guedes treated Alan with heparin at 2:45 PM on November 24th, at a dose level of 2 cc (50% concentration of 1000 IU/ml) per hour by intravenous infusion. The baby’s weight was 4.57 kg and the estimated heparin dose was 219 IU/kg per hour. Based on this dose, the estimated total dose of heparin infused up to the time of the CT scan (five hours) was 1095 IU/kg, which is about 8.8 times the recommended maintenance dose for infants of 125 IU/kg per five hours, recommended by the Physicians’ Desk Reference [17]. Hemorrhage can occur virtually at any site in patients receiving heparin. Patients suffering from anemia, any unexplained symptoms, and/or having low blood pressure are at the greatest risk of having serious hemorrhagic events following a therapeutic dose of heparin. Alan had hypotension, and his hematocrit was very low (25.3%). In addition, the baby was treated with adenosine, which is a potent vasodilator in most vascular beds, causing significant hypotension
(Table 7, Table 11). Heparin also induces the formation of white clot due to the aggregation of platelets. At 3:15 PM, at about 30 minutes post heparin infusion, blood analysis showed increased prothrombin time and fibrinogen split product (Table 9). The treatment with heparin explains the bleeding seen on the CT scan taken at 7:50 PM on November 24th and the fall in the platelet count seen on November 25th (Table 12).


Furthermore, the bleeding in the brain was made worse by Alan’s second treatment with heparin at 8:00 AM on November 25th. The baby was given heparin by infusion similar to the dose given the day before (described above, 219 IU/kg per hour). This treatment was not justified at all, because heparin at a high therapeutic dose should not be given to any patient suffering from bleeding and hypotension [17]. Baby Alan had a bleeding gastric ulcer, subdural hemorrhage, bleeding in the brain, and hypotension. The platelet count prior to the administration of heparin on November 24th was 571,000/µL of blood and dropped to 397,000/µL (30% reduction) at 5:45 AM on November 25, 1997 (at about 15 hours following the start of the first heparin infusion). Heparin increases the tendency of the platelets to aggregate and form a clot. Blood analysis values for November 24th through November 27th are presented in Table 12. Alan also suffered from disseminated intravascular coagulation (DIC) as a result of his treatment with heparin. At 3:15 PM, at about 30 minutes post heparin infusion, blood analysis showed increases in fibrinogen split product (160 µg/mL) and prothrombin time (11.6 seconds). These values are 1600 % and 115% of normal respectively, and they returned to normal on November 26th following the cessation of the treatment with heparin.

To make matters even worse, Dr. Guedes treated the baby with excessive amount of sodium bicarbonate by IV to treat acidosis. This treatment caused severe metabolic alkalosis, and the blood pH reached a very critical high level of 7.7. This treatment caused severe hypoxia by preventing the hemoglobin from releasing oxygen to the tissues; it caused reduced potassium level in the blood, and caused cerebral edema, as explained in sections III and IV of this report. Alan’s blood pH was 7.1 and serum potassium level was 4.9 mEq/L on November 24th. The potassium dropped to critical low of 2.3 mEq/L at 5:45 AM on 11/25 following the treatment with excessive amount of sodium bicarbonate. The blood pH was at critically high levels of 7.6-7.7.


Dehydration, polyurea, weight loss, and wasting are symptoms and complications of diabetes mellitus. In the first twenty-four hours, baby Alan’s input was 725.8 mL, while his output during this period was 786 mL. Net output was 60.2 mL. On November 24th, Alan weighed 10.05 pounds, and on November 29th, 9.0 lb. He lost 1.05 lb (10% of his weight) in five days during his hospital stay despite treatment with relatively high volume of fluid IV. Furthermore, his average serum creatinine on November 24th was 0.45 mg/dL (75% of low normal value) and dropped to 0.2 mg/dL (33% of low normal) on November 27th (Table 12). Low creatinine is an indicator of low muscle mass and wasting disease.


In addition, the baby was treated with an antidiuretic hormone (DDAVP) on November 28th to prevent dehydration (Table 13). DDAVP is a synthetic analog of the natural pituitary hormone 8-arginine vasopressin (ADH), an antidiuretic hormone affecting renal conservation. This fact shows that he was responding to medication in spite of being declared brain dead on November 27, 1997. Also, he was able to maintain his body temperature at about normal levels following his treatment with antibiotics. The treatment with antibiotics reduced the blood white blood cell count from 20, 900 to 13, 600/µL and body temperature from 105.8 F to 99.8 F. This indicates that his fever was caused by bacterial infections and not by brain injury, as Guedes claimed.


Dr. Guedes and Florida Hospital contacted the Orange County Sheriff’s Office and the Child Protective Office on November 24, 1997 and filed a report of child abuse based on the assumption that baby Alan was injured as a result of abuse by his father. Alan R. Yurko was arrested by officers from the Orange County Sheriff’s Office on November 26, 1997, while his son Alan was still alive in Florida Hospital. I found that the actions of Dr. Guedes and Florida Hospital are not supported by any medical facts. Their actions are tragic for Alan’s family and dangerous for the entire society!


Dr. Guedes assumed that Mr. Yurko was guilty of child abuse, yet his own examination of baby Alan revealed no injuries caused by trauma. Also, he did not wait to the end to see the result of the autopsy. In addition, he did not review the adverse reactions of medications that he gave to the baby on November 24th to see if the heparin and other medications had something to do with bleeding in the brain. Then he gave more heparin and sodium bicarbonate the next morning, which caused more bleeding. On November 24th, the subdural bleeding was on the right side of the brain only, and on November 29th, the bleeding was on the right and left sides of the brain. Guedes gave excessive amount of heparin and sodium bicarbonate, which can cause bleeding in any child who has hypotension, anemia, and tissue inflammation similar to baby Alan’s. The medical practice and action of Dr. Guedes make no ill child safe from bleeding in the brain and other tissues. Also, no parent can be safe from being accused of killing his or her child by shaking. His work should be investigated to stop these horrible tragedies from happening to people!

V-B. Review of Dr. Matthew A. Seibel’s testimony
Dr. Matthew Seibel is a general pediatrician and consultant with the child protection team based in the children’s hospital in Orlando. He examined baby Alan and reviewed his chart on November 25th for about 30 to 45 minutes in Florida Hospital, and declared that the baby was injured as a result of child abuse. He gave his conclusion about the cause of injuries without review of Francine’s medical records while pregnant with Alan, the baby’s medical records from birth until his hospitalization on November 24, 1997, the adverse reactions to the vaccines and medications given to baby Alan, or the autopsy findings. These issues and their impact on this case are explained in sections I through IV of this report. Below are the questions presented to Dr. Seibel in court by the State of Florida and defense attorneys, and his answers to these questions [13]. They clearly show that he presented his conclusion based on a theory only and not based on the medical evidence.

1. Examination and diagnosis:
The state: Where did you first examine the child?
Seibel: The one and only one time I examined him was in pediatric intensive care unit at Florida South on November 1997.

The state: And once you examined him and reviewed his medical chart, did you have a diagnosis?
Seibel: Yes

The state: What was that?
Seibel: The child had received inflicted trauma that was seen as both brain injuries as well as injuries to several ribs that is characteristic of an abusive situation.

The state: As part of your examination of this child, would you rule out any natural disease and causes in making your diagnosis?
Seibel: You always like to try to put a nice face on it and you look as hard as you can to make sense of it, but I was not able to find any natural diseases other than trauma to cause these injuries in this child.

Defense attorney: You talk about a few minutes, examining the child, a few minutes looking at the x-rays, a few minutes reading the report. Totally, how much time you spend examining this case?
Seibel: Probably there or at the hospital between a half hour and 45 minutes.

Defense attorney: Did you make any attempts to get this child’s prior medical records, prenatal record, anything like that.
Seibel: No sir, I did not .

Defense attorney: Did you look up the medical Examiner’s report?
Seibel: I have been made aware of it. I do not have it with me. I do not remember reviewing it very, very carefully.


2. Diffuse axonal injury:
Defense attorney: In your report you talk about diffuse axonal injury; is that correct?
Seibel: Yes, Sir, that is the current theories regarding these children when there is significant bleeding in the brain as we saw in this case.

Defense attorney:
We are not talking about theories. In your examination of this child you actually did not see diffuse axonal injury?
Seibel: No Sir, I did not.


3. Retinal hemorrhage:
Defense attorney: Do you know how much hemorrhage was found in the child’s retina in this case?
Seibel: No, I do not.

Defense attorney: Do you know whether or not there was retinal hemorrhage in both eyes or one eye?
Seibel: I am not sure which one or who did it.


4. Rib fracture:
The state: Are there fractures that can be the result of birth trauma?
Seibel: Yes

The state: And what type of fractures would that be?
Seibel: The most common fractures that we see is fractures of the clavicle which is the collar bone right here.

The state: Within a reasonable degree of medical certainty, rib fractures are considered to be abuse?
Seibel:l: Yes

Dr. Seibel’s answers to the questions in court said it all. He spent only 30 to 45 minutes on the entire case. He did not see axonal injury. He did not know about the bleeding in the retina. He did not evaluate the child’s and mother’s medical records. He gave his conclusion about the cause of injury on November 25th when the baby was still alive. He said that the child had several rib fractures, but the medical records showed the baby had only rib #6 fracture. Furthermore, my review of the medical literature revealed that multiple rib fractures do occur during labor. I presented the findings of four studies that show the occurrence of ribs fracture during labor in Section IV-J of this report. Also, I presented a study that showed fracture of long bones during labor. Seibel stated that the child’s injuries were caused by trauma, but the examination by Dr. Guedes on November 24th revealed no injuries due to trauma on the head or the body of the baby, except for a minor bruise under the right eyelid.


It is very hard to imagine that Dr. Seibel testified in a court on this very serious matter based solely on a theory; without evaluating the medical evidence and the related published medical literature. His testimony put an innocent father in prison for life and destroyed the family’s structure. I believe that his unscientific and unjustifiable actions resulted in a tragedy which puts our society in danger. His actions should be investigated.


As previously explained in this report (sections III, IV), the bleeding in the brain and other tissues in baby Alan resulted from his treatment with excessive amounts of heparin and sodium bicarbonate in the hospital. I reached my conclusions after spending more than two hundred and fifty hours evaluating the medical evidence and the related published medical information. Baby Alan’s case and similar cases are very complicated medical matters, as shown by the information presented in this report. They should be evaluated by using differential diagnosis prior to giving any conclusion. I am certain that spending only 30 or 45 minutes on this case, as Dr. Seibel did, could not lead to a valid conclusion. Physicians should take all facts in to consideration when they are faced with a complicated task such as this one.

V-C. Review of Dr. Gary Steven Pearl’s testimony
Dr. Pearl is a consultant neuropathologist working with the Medical Examiner’s office. He examined the brain, spinal cord, and the eyes of baby Alan grossly on December 19, 1997, and also prepared slides. His pathology findings on the brain, spinal cord, and the right eye do not support the medical examiner and other physicians’ claims that the bleeding in the brain occurred in minutes or in a few seconds. Dr. Pearl examined the hematoma in the subdural space of the brain and spinal cord and observed the proliferation of fibroblasts in layers, clotted blood with no fibrosis, and fresh blood. He estimated the age of the oldest portion of the subdural hematoma to be two to five days [13]. This means that the blood was released in at least three stages, as described in Section IV-D of this report, and this contradicts the theory of the other state witnesses that the bleeding in the brain occurred in minutes or in a few seconds.

Dr. Pearl also stated that baby Alan suffered from disseminated intravascular coagulation (DIC), which led to the hemorrhage in the subdural space, and that the DIC was induced by brain injury. I agree with Dr. Pearl that the baby suffered from DIC, as shown clinically (Table 9, Table 12); but the reason for the DIC was the treatment with excessive doses of heparin, as explained in this report (Sections III, IV). Dr. Pearl missed the role of heparin in the bleeding in the brain and spinal cord and in the formation of DIC because he did not review the baby’s records and treatment chart during his five days in the hospital.


Furthermore, Dr. Pearl observed swollen blood vessels and acute degeneration of nerve cells in the brain and spinal cord and stated that these are signs of acute injuries. They occurred as a response to a brain injury. I agree with Dr. Pearl that these lesions are signs of acute injuries occurring within 2-5 days prior to autopsy, but these lesions were induced by severe hypoxia caused by excessive treatment with sodium bicarbonate on November 24th and 25th, by severe anemia, apnea, cardiac arrest, and hypotension, as explain in this report (Sections III, IV). The treatment with bicarbonate caused severe metabolic alkalosis. The blood pH was 7.7. This prevents the hemoglobin from releasing oxygen to the tissues, and it caused hypoxia and cerebral edema in this case. Brain edema was confirmed at autopsy. Dr. Pearl missed the influence of these factors on the formation of the lesions in the brain and the spinal cord because he did not review Alan’s medical records.


Dr. Pearl also observed meningeal inflammation and called it hemogenic meningitis (inflammation of the meninges resulting from the presence of blood). He based his conclusion on the quantity and type of inflammatory cells present in the inflamed tissue. This lesion lacked the presence of neutrophils. I believe that Dr. Pearl’s interpretation of the tissue changes is incomplete because he did not review the baby’s medical records. The baby was treated with high therapeutic doses of three types of antibiotics, and this treatment reduced the severity of the acute inflammation observed at the time of autopsy. The swollen blood vessels, the presence of edema in the brain, fever (105.8 F) and the elevated white blood cell count (20, 900/µL) observed on November 24th (Table 12) indicate that the baby suffered from acute meningitis. However, the severity of the acute inflammation in the tissue was reduced by the treatment with high therapeutic doses of gentamicin, rocephin, and Claforan (cefotaxime sodium) on November 24th
(Table 6, Table 7). The treatment with antibiotics reduced the blood white blood cell counts from 20, 900 to 13, 600/µL and body temperature from 105.8 F to 99.8 F.


Also, Dr. Pearl did not present evidence in court that showed diffuse axonal injury. In addition, Dr. Gore's autopsy report makes no mention of Dr. Pearl’s pathology findings on the brain, spinal cord, and eyes. Other state witnesses also did not present any evidence in court that showed diffuse axonal injury. However, they claimed that diffuse axonal injury is characteristic of “Shaken Baby Syndrome” (SBS). Hemorrhage in the retina is another claimed characteristic marker for SBS. Dr. Pearl stated that the bleeding in the right retina was very minor and did not impact his opinion in this case. Below are the defense attorney’s questions presented to Dr. Pearl in court regarding the axonal injury and the retinal bleeding, and his answers to these questions.

1. Diffuse axonal injury:
Defense attorney: Now, I believe you testified that there was some slides that shows the diffuse axonal injuries?
Pearl: That is correct.

Defense attorney: Do you have those slides?
Pearl: I do not have any of the slides.

Defense attorney: But there were slides that was done that have shown the diffuse axonal injury?
Pearl: There is a glass slide that does. I did not photograph that.


2. Bleeding in the retina of the right eye:
Defense attorney: Was there hemorrhage in the retina or behind the retina?
Pearl: There was a small hemorrhage.

Defense attorney: Minute?
Pearl: Minute.

Defense attorney: Was that significant?
Pearl: That wasn’t significant to me. Really did not impact on my evaluation of the case.

Furthermore, Dr. Pearl did not review Francine’s medical records during her pregnancy with Alan, the baby’s medical records from birth until his hospitalization on November 24, 1997, or the adverse reactions of vaccines and medications given to baby Alan. This limited his ability to interpret his pathology findings and to provide valid conclusions about the causes of injury and death.

V-D. Review of Dr. Douglas R. Shanklin’s testimony
Dr. Shanklin, the defense witness evaluated the H & E stained tissue sections of the brain and other organs and stated that baby Alan died from natural causes. I agree with Dr. Shanklin that baby Alan suffered from health problems at birth and following birth. These included respiratory distress syndrome, jaundice, and growth retardation. However, these problems did not cause his cardiac arrest or the bleeding in the brain and other tissues observed in the autopsy. Alan’s cardiac arrest was caused by hypokalemia developed as a result of metabolic and respiratory acidosis due to diabetes and pneumonia. Alan’s health problems at birth and following birth increased his susceptibility to develop adverse reactions to vaccines. The six vaccines that he received on November 11, 1997 caused his infections and diabetes, as explained in sections II and IV of this report. The bleeding in the brain and other tissues was caused by the excessive treatment with heparin and sodium bicarbonate given in the hospital, as explained previously.

Dr. Shanklin made very important contributions to this case. He stated that baby Alan’s kidneys were not fully developed. This finding may explain the mother’s problem with oligohydramnios, as explained in section II of this report. He also stated that the baby suffered from pneumonia and meningitis in the brain region and the spinal cord. His findings may explain the susceptibility of these regions to bleeding caused by the treatment with heparin and sodium bicarbonate. The inflammation in these regions affected the integrity of the blood vessels and caused predisposition to the leakage of fluid and blood induced by treatment with excessive doses of heparin and sodium bicarbonate. In addition, he described old neurological injuries in the brain and spinal cord. I believe that the high levels of bilirubin observed in the first week following birth caused these injuries (Table 3). I addressed this issue in section II.

V-E. Analysis of the state’s claim
During Alan R. Yurko’s jury trial, the prosecutor, Ms. Wilkinson, presented only one theory—that Baby Alan died as a result of “Shaken Baby Syndrome” (SBS), and Mr. Yurko, the father of the child, did it. My review of the medical evidence and the trial transcript revealed that the prosecutor did not prove her case that the injuries were caused by trauma and that Mr. Yurko abused his child. However, the prosecutor achieved her goal of convicting Mr. Yurko of a horrible crime he did not commit. He received a life sentence + 10 years. I believe that the prosecutor used unfair practices and violated Mr. Yurko’s right of getting a fair trial. Below is a list of the prosecutor’s unfair tactics and evidence that shows the state did not prove its case.

1. The prosecutor presented trauma as the only possibility for the cause of injuries in this case; but Dr. Guedes examined the baby on November 24th, and his examination did not reveal any injury caused by trauma, except for the minor bruise under the right eyelid. In addition, Mr. Yurko never stated that he abused his child, and no one observed him hurting baby Alan.
2. The prosecutor did not discuss the adverse reactions of vaccines given to baby Alan (Table 5). The studies presented in section II of this report clearly show that these vaccines caused severe adverse reactions, such as apnea, cardiac problem, and respiratory infections, in premature infants. Baby Alan developed diabetes resulting from an infection and complications of his vaccination. This led to his cardiac arrest on November 24, 1997.
3. The prosecutor did not discuss the adverse reactions of medications given to Alan on November 24th through November 29th. It is very obvious that heparin causes bleeding at the doses given.
4. The prosecutor stated that the baby developed subdural bleeding in the brain, retinal bleeding, and diffuse axonal injury as a result of SBS. However, the autopsy and the pathology findings clearly show that the subdural bleeding did not occur in a few minutes as the SBS theory claimed. In addition, none of the state witnesses presented evidence in court that they found diffused axonal injury. Also, the bleeding in the retina of the right eye was very minor. Even Dr. Pearl stated that this injury was very minute and had no impact on his evaluation.
5. The prosecutor did not ask Dr. Gore to provide the x-ray findings to prove that Alan had fractures in ribs 5, 7, and 10. He stated in his autopsy report that he used an x-ray to confirm his findings. In addition, the prosecutor held the x-ray findings taken on September 16, 17, and 18 as proof that the rib fractures did not happen during labor. It takes at least 7 days for the calluses to form, and on September 18, 1997, the baby was 3 days old. However, the prosecutor did not accept the x-ray findings of November 24th and 25th that the baby had only rib # 6 fracture. She stated that the fractures are better seen at autopsy, but she overlooked the fact that Dr. Gore stated in his report that he took the x-ray to confirm the rib fractures. It seems that the prosecutor was trying to select certain evidence to support her theory that the father was abusing his son and caused multiple rib fractures. Her actions showed that she did not conduct her business in a fair and impartial manner.
6. The prosecutor allowed Dr. Gore to present in court as evidence of trauma two photographs of minor contusions in the temporal areas of the head that occurred in the hospital about one day prior to autopsy. The medical examiner’s main objective should be discussing the causes of injuries that caused death in this case. I believe he used these photos to influence the jury’s thinking that physical force was used, and the prosecutor should not have allowed this.
7. None of the state witnesses evaluated all of the evidence relevant to this case, such as prenatal records and the baby’s medical records from birth until the time of his
cardiac arrest, and the prosecutor did not question the validity of their testimonies.

 

Section VI. Conclusions and Recommendations

Baby Alan Ream Yurko suffered from several serious heath problems at birth and following birth, such as respiratory distress syndrome, hypoglycemia, jaundice, growth retardation, and bacterial infections. His serum bilirubin level was 17.4 mg/dL at 3 days post-birth, which is capable of causing encephalopathy. Also, his risk of developing encephalopathy and of suffering from hypoxia was increased by the treatment with antibiotics that bind with albumin and release bilirubin. In addition, baby Alan had high risk of developing congenital deformity of organs and the skeleton because his mother suffered from gestational diabetes and oligohydramnios during the pregnancy. His growth rate in the first month of life was poor, but he showed a good improvement in his growth rate during the second month.

Unfortunately, his pediatrician gave him six vaccines at two months of age and sent him home without monitoring or medical supervision. It has been reported that premature children, such as baby Alan, who were vaccinated prior to 70 days of age showed very high risk of developing serious health problems, such as apnea, bradycardia, and oxygen desaturation, that required medical intervention. Alan developed fever, reduction in food intake, lethargy, and became irritable a few days following vaccination. The medical evidence indicates that his vaccination induced these symptoms.


At thirteen days post vaccination, the baby had cardiac arrest and apnea. The clinical tests revealed that he was suffering from diabetes mellitus and the complications of diabetes, such as bacterial infections, gastric ulcer, metabolic acidosis, hypokalemia, dehydration, anemia, weight loss, loss of muscle tone, and cardiac arrest. The six vaccines received on November 11, 1997 were the likely cause of his infections and diabetes (Table 5). His cardiac arrest and apnea on November 24th, resulted from hypokalemia which was a result of metabolic and respiratory acidosis. It appears that his diabetes was induced by bacterial infections, because his blood sugar level of 397 mg/dL on November 24th returned to normal two days following IV antibiotics. He also had pneumonia, meningitis, eye infection, and liver and heart damage. His liver serum enzymes and LDH levels were also reduced significantly following the treatment with antibiotics, and this indicates that he had liver, and maybe heart, bacterial infections (Table 12).


Unfortunately, at Florida Hospital baby Alan was treated on November 24 and 25, 1997 with high doses of sodium bicarbonate that caused metabolic alkalosis (pH 7.7) and severe hypoxia. He was also treated with high doses of heparin that caused bleeding in tissues. Heparin caused severe subdural bleeding in the brain and spinal cord, minor bleeding in the brain and the retina of the right eye, and bleeding in the lungs. It also caused disseminated intravascular coagulation (DIC). I am very surprised to see that Dr. Guedes treated Alan with high doses of sodium bicarbonate and heparin at 8:00 AM on November 25th despite the fact that he was suffering from severe metabolic alkalosis (PH 7.61-7.7) and bleeding in the brain and subdural space
(Table 8, Table 11). Treatments with high doses of sodium bicarbonate and heparin were not medically justified.


The treatment with bicarbonate for individuals with diabetes should stop at pH 7.2, and this treatment carries high risk of causing cerebral edema, as happened in this case. Heparin should not be given to patients suffering from bleeding, hypotension, and anemia, because of the high risk of bleeding in such patients associated with heparin. Baby Alan had hypotension, anemia, hypoxia, bacterial infections in several organs, bleeding gastric ulcer, and bleeding in several organs. It seems that Dr. Guedes overlooked the adverse reactions of sodium bicarbonate and heparin, as well as the recommendations presented in the PDR and medical textbooks concerning the treatment with these agents.


The radiology findings of November 24 and 25, 1997 showed that Alan had only rib #6 fracture. Rib fractures have been observed to occur during labor even in mature babies. Also, oligohydramnios can cause positional skeletal deformity. The medical evidence indicates that the fracture of rib #6 most likely happened during labor. The claim of Dr. Gore that the baby had additional ribs fractures is not valid, because he did not provide any evidence to support his claim, such as x-ray results and films. In addition, there were no calluses observed in the cut sections of the swollen cartilage at autopsy.
Dr. Gore’s autopsy report and his court testimony related to this case suffer from accuracy problems and contain very serious contradictions. They are certainly not reliable medical evidence to be used to answer questions about the cause(s) of death. He stated that the heart was normal and described the histology of the heart, but in the same report, he mentioned that the heart was donated prior to his examination. This indicates that Gore was describing the heart of a different child and got his cases mixed up. This is very serious, and his work should be investigated. Also, he stated in his report that the cerebrospinal fluid was clear; whereas, in court he said that the CSF fluid was mixed with blood. This is another serious problem found in Gore’s work.


In addition, Gore said in court that the baby did not have meningitis, but his report does not contain any description of the histology of the meninges to show that he examined them. The other two pathologists who evaluated the slides of the meninges observed acute changes that indicate meningeal inflammation. Furthermore, he reported that the baby’s head circumference was 22 cm, and that is obviously wrong. It was 37.5 cm eighteen days prior to the autopsy date.


Furthermore, Gore did not provide the medical evidence in his report and in court to support his conclusion that baby Alan died as a result of “Shaken Baby Syndrome.” Gore stated that the subdural bleeding in the brain occurred in a few minutes or even in a few seconds due to vigorous shaking. However, the autopsy and the pathology findings showed that the bleeding occurred during 2-5 days and at least in three stages. The bleeding also occurred in the spinal cord and the lungs, which has nothing to do with the SBS theory, but it indicates that the bleeding resulted from cardiovascular problems. In addition, he did not show any evidence that the baby had diffuse axonal injury in the brain. No description of axonal injury was presented in his report, and he did not show a single slide describing such a lesion during the trial. The other three state witnesses also did not show any slide or picture of an axonal injury.
Furthermore, I find Gore’s conclusion about the cause of death invalid, because he did not review the baby’s prenatal records, the mother’s records during her pregnancy with Alan, or adverse reactions of vaccines and medications given to the baby. He missed the facts that the baby had diabetes, hypokalemia, anemia, metabolic alkalosis, bleeding as a result of heparin treatment, and hypoxia and cerebral edema due to the treatment with bicarbonate.


In addition, my review of the medical literature revealed that axonal injury in the brain and spinal cord can occur in cases of cardiac arrest, edema, hypoglycemia, and from other causes; and it is not necessarily a characteristic lesion of injuries caused by trauma. Also, treatment with high doses of heparin and sodium bicarbonate can induce bleeding in cases of hypotension, anemia, and inflammation in tissues, as happened in this case. Heparin and sodium bicarbonate are commonly-used agents to treat clotting disorder and acidosis. Therefore, cases of SBS should be investigated to rule out the contribution of therapeutic agents and other factors in the pathogenesis of brain lesions. I believe that cases of individuals who were convicted of killing babies by SBS based on the axonal injury, subdural bleeding, and eye bleeding should be reexamined.


Also, I believe that Gore’s action of presenting in court two photographs of minor contusions that occurred in the hospital, which were unrelated to the cause of death, is scientifically and professionally unjustified. I believe that he did it to influence the thinking of the jury that physical force had been used on the baby.


My review of the evidence indicates that Dr. Guedes’ testimony in court is unsupported by science and medical facts. He did not review the medical evidence related to this case such as prenatal record, the baby medical record, and the pathology findings. In addition, he did not review related published literature describing the adverse reactions to vaccines and medications given in this case. Also he did not reveal to the court that the baby was treated with three types of antibiotics to fight infections, bicarbonate and heparin; and that the baby suffered from diabetes, dehydration, hypokalemia and complications of diabetes. I believe that withholding of these vital data from the court resulted in a negative outcome of the trial, and this issue should also be investigated.


Dr. Guedes’ treatment of baby Alan with high doses of bicarbonate and heparin caused serious injuries and death and should be investigated. In addition, this treatment produces bleeding in the brain and other tissues and axonal injuries, which are considered by the proponent of SBS as characteristic markers for it. It led to the false conviction and the imprisonment of Mr. Yurko.


I believe that Dr. Seibel’s testimony in court was also unsupported by medical evidence and science, based on the facts that he did not review the medical evidence that related to this case such as prenatal record, the baby medical record and the related published literature describing the adverse reactions to vaccines and medications given, and the pathology findings. He spent only 30-40 minutes on this case, and this certainly did not give him the expertise to testify on this complicated medical case.


Alan Yurko and his family suffered two tragedies as a result of problems with our current medical system regarding the vaccination of premature babies, the treatment given to baby Alan in the hospital, and the approaches of the state witnesses in evaluating the evidence in this case. The first tragedy is the loss of baby Alan because of adverse reactions to vaccines and the excessive doses of heparin and sodium bicarbonate given in the hospital. It seems that his pediatrician was unfamiliar with the adverse reactions of vaccines in premature children. He gave him six vaccines and sent him home. Dr. Guedes also overlooked the adverse potentials of heparin and bicarbonate. These issues should be addressed by the state and medical authorities, which might save babies from death due to adverse reactions to vaccines and treatment with the wrong medications.


The Yurkos’ second tragedy was the conviction of Mr. Yurko with a horrible crime he did not commit. He was convicted because the state’s four expert witnesses did not take the time to review the evidence and the related published literature. Nor did they sort out the facts, so that their testimonies were based on a theory only. The prosecutor contributed to the problem by focusing on only one theory. She also allowed Gore to present evidence that had no connection to the case such, as the photographs describing the two contusions on the baby’s head.


I believe that the state of Florida has the responsibility to review the evidence presented in this report. It shows that Mr. Yurko is innocent, and they should take speedy action to free him from prison. Also, the state should investigate the involvement of the state witnesses and the prosecutor with similar cases resulting in the conviction of parents accused of killing their children by SBS. Furthermore, I believe that the state and the doctors who caused the Yurko’s tragedy should compensate Mr. Yurko and his family for the loss of their child, their suffering, and the expenses paid.

The objective of the state and the medical system should be determining the factual causes that led to the illness and death of a child and to prevent such problems from happening to other children. Accusing innocent parents of abusing and killing their children based on unsupported theory, as it happened in this case, will not prevent the death of other children by vaccines and incorrect medications. But it certainly puts innocent people in prison and causes suffering. It also costs the taxpayers huge sum of money to pay for trials and legal fees. I spent more than 250 hours working on the Yurko case to find the factual causes of death and to write the detailed report. I hope that the state of Florida, the medical system, and our society will take advantage of this opportunity to see the real problems facing a premature baby who received vaccines and was not properly monitored by the medical community. This tragedy affects not only the child, but also its family and the rest of the world.

TOC

References

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[34] Shannon P, Smith CR, Deck J, Ang LC, Ho M, Becker L. Axonal injury and the neuropathology of Shaken Baby Syndrome. Acta Neuropathol (Berl).
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[35] Hartmann RW Jr. Radiological case of the month. Rib fractures produced by birth trauma. Arch Pediatr Adolesc Med 1997; 151(9):947-8.
[36] Rizzolo PJ, Coleman PR. Neonatal rib fracture: birth trauma or child abuse? J Fam Pract 1989; 29(5):561-3.
[37] Cumming WA. Neonatal skeletal fractures. Birth trauma or child abuse? J Can Assoc Radiol 1979; 30 (1):30-3.

TOC

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